Glucose-6-phosphate dehydrogenase deficiency is genetic?

Patient's question:

G6PD deficiency in infants is a genetic factor or cause? Are there other factors or causes of this disease? Is G6PD deficiency a genetic factor or cause? Are there other factors or causes of this disease? Can this disease only be prevented? Can it be treated? Does blood testing during neonatal jaundice affect this result?

Doctor's answer:

A lack of glucose-6-phosphate dehydrogenase (G6PD) in red blood cells is called G6PD deficiency (commonly known as favism). This is a hereditary disease prevalent in southern and southwestern provinces. In Guangdong Province, 5-10% of the population suffers from this disease. Prevention is key for favism. Individuals or families with a history of favism should avoid eating broad beans and avoid contact with broad bean pollen. Medications such as primaquine (an antimalarial drug), chloramphenicol and sulfonamides (antibiotics), aspirin (a fever-reducing and pain-relieving drug), and commonly used mothballs (naphthalene) in daily life should not be used.

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